circRNA basic information
circBase ID: -
Name: hsa_circ_MTO1
Synonym: hsa_circRNA_104135
Host Gene: MTO1
Genomic location(hg19): -
Genomic location(hg38): -
Subcellular localization: not tested
 
 
 
 
 
 
 
Disease basic information
MONDO ID:
0002691
MONDO name: liver cancer
Disease details: liver cancer
Disease DO ID:
3571
Disease MeSH ID:
-
Disease NCIt ID:
C34803
Disease ICD11 ID:
1605020868
Disease OMIM ID:
-
Species: Human
Species details: Homo sapiens
Tissue specimen:

liver tumor tissues; adjacent normal tissues

Cell lines:

Huh7; Hep3B; MHCC-97L; MHCC-97H; SMMC-7721; HepG2; L-02

In vivo animal model:

-

circRNA-disease information
Expression pattern:
DN
Associated gene: NOX4
Associated microRNA: miR-9-5p
Biological function: Inhibits proliferation and migration and promotes apoptosis of hepatoma cells (tumor suppressor role).
Molecular mechanism: Acts as a molecular sponge of miR-9-5p, relieving miR-9-5p-mediated repression to upregulate NOX4.
Biological pathway or process:

proliferation (inhibits); migration (inhibits); apoptosis (promotes); ceRNA regulation (promotes)

Detected method:
Q
M
Validation methods:

Microarray; RT-qPCR; Luciferase Reporter Assay; Transfection; CCK8; Transwell Assay; Annexin V/PI Flow Cytometry; Western Blot; IHC (Immunohistochemistry); Clinical Sample Validation; Bioinformatics Analysis

Clinical significance:

circMTO1 is a potential diagnostic biomarker and therapeutic target for HCC.

Description:

circMTO1 is down-regulated in HCC tissues/cell lines and functions as a tumor suppressor by inhibiting proliferation/migration and promoting apoptosis. Mechanistically, it sponges miR-9-5p to upregulate NOX4, forming a circMTO1/miR-9-5p/NOX4 ceRNA axis.

Confidence score:

0.7357

Other information
Title:

Mechanism of the Regulatory Effect of Overexpression of circMTO1 on Proliferation and Apoptosis of Hepatoma Cells via miR-9-5p/NOX4 Axis.

Journal: Cancer management and research
Published: 2020
PubMed ID: 32547227
Study type:

combined biological and clinical study

Data availability: -
Code availability: -