circRNA basic information
circBase ID: hsa_circ_0001696
Name: hsa_circ_HERPUD2
Synonym: -
Host Gene: HERPUD2
Genomic location(hg19): chr7:35707043-35712888:-
Genomic location(hg38): chr7:35667433-35673278:-
Subcellular localization: not tested
 
 
 
 
 
 
 
Disease basic information
MONDO ID:
0005575
MONDO name: colorectal cancer
Disease details: colorectal cancer
Disease DO ID:
5672, 9256
Disease MeSH ID:
-
Disease NCIt ID:
C4978
Disease ICD11 ID:
-
Disease OMIM ID:
114500
Species: Human
Species details: Homo sapiens
Tissue specimen:

CRC tissues; adjacent normal tissues

Cell lines:

HCT116; SW620

In vivo animal model:

-

circRNA-disease information
Expression pattern:
DN
Associated gene: CDK4, cyclin D, cyclin E, MMP9
Associated microRNA: -
Biological function: Inhibits colorectal cancer cell proliferation and migration; knockdown promotes colony formation.
Molecular mechanism: Knockdown increases CDK4, cyclin D, cyclin E and MMP9 (mRNA/protein), suggesting regulation of cell-cycle and migration-related factors; ceRNA/miRNA and protein-binding mechanisms were not assessed.
Biological pathway or process:

proliferation (inhibits); migration (inhibits); cell cycle (inhibits)

Detected method:
Q
Validation methods:

RT-qPCR; Transfection; CCK8; Colony Formation Assay; Wound Healing Assay; Western Blot; Clinical Sample Validation

Clinical significance:

May act as a potential novel molecular diagnostic marker for CRC (limited by small sample size).

Description:

hsa_circ_0001696 is down-regulated in colorectal cancer tissues. Functionally, its knockdown promotes CRC cell proliferation, colony formation, and migration, accompanied by increased CDK4/cyclin D/cyclin E and MMP9 levels, suggesting a tumor-suppressive role.

Confidence score:

0.5414

Other information
Title:

Hsa_circ_0001696 modulates cell proliferation and migration in colorectal cancer.

Journal: Oncology letters
Published: 2021
PubMed ID: 33552272
Study type:

combined biological and clinical study

Data availability: The datasets generated and/or analyzed during the present study are available from the corresponding author upon reasonable request.
Code availability: -