circRNA basic information
circBase ID: hsa_circ_0001174
Name: -
Synonym: circ_0001174
Host Gene: -
Genomic location(hg19): chr20:58755107-58755971:+
Genomic location(hg38): chr20:60180049-60180913:+
Subcellular localization: not tested
 
 
 
 
 
 
 
Disease basic information
MONDO ID:
0009807
MONDO name: osteosarcoma
Disease details: osteosarcoma / OS
Disease DO ID:
3347
Disease MeSH ID:
D012516
Disease NCIt ID:
C9145
Disease ICD11 ID:
-
Disease OMIM ID:
-
Species: Human
Species details: Homo sapiens
Tissue specimen:

OS tissues; adjacent tissues / paracancerous tissues

Cell lines:

MG63; HOS; U2OS; hFOB1.19

In vivo animal model:

-

circRNA-disease information
Expression pattern:
UP
Associated gene: MACC1
Associated microRNA: miR-186-5p
Biological function: promotes osteosarcoma cell proliferation, migration, and invasion
Molecular mechanism: ceRNA sponging miR-186-5p to upregulate MACC1
Biological pathway or process:

proliferation (promotes); migration (promotes); invasion (promotes); ceRNA regulation (promotes)

Detected method:
Q
S
Validation methods:

RNA-seq; RT-qPCR; Clinical Sample Validation; Transfection; CCK8; Colony Formation Assay; Transwell Assay; Wound Healing Assay; Western Blot; Bioinformatics Analysis; Survival Analysis; Luciferase Reporter Assay

Clinical significance:

High circ_0001174 expression was closely associated with shorter disease-free survival (DFS).

Description:

circ_0001174 is up-regulated in osteosarcoma tissues and cell lines and promotes OS cell proliferation, migration, and invasion. It acts as a ceRNA by sponging miR-186-5p, thereby increasing MACC1 expression and associating with shorter disease-free survival.

Confidence score:

0.7369

Other information
Title:

Circ_0001174 facilitates osteosarcoma cell proliferation, migration, and invasion by targeting the miR-186-5p/MACC1 axis.

Journal: Journal of orthopaedic surgery and research
Published: 2022
PubMed ID: 35279159
Study type:

combined biological and clinical study

Data availability: available from the corresponding author upon request
Code availability: -