circRNA basic information
circBase ID: -
Name: hsa_circ_MTO1
Synonym: circRNA-MTO1 / hsa-circRNA-007874
Host Gene: MTO1
Genomic location(hg19): -
Genomic location(hg38): -
Subcellular localization: nucleus and cytoplasm
 
 
 
 
 
 
 
Disease basic information
MONDO ID:
0007254
MONDO name: breast cancer
Disease details: breast cancer / BC
Disease DO ID:
1612
Disease MeSH ID:
-
Disease NCIt ID:
C9335
Disease ICD11 ID:
1047754165
Disease OMIM ID:
-
Species: Human
Species details: Homo sapiens
Tissue specimen:

-

Cell lines:

MDA-MB-231; MCF-7; MDA-MB-453; SKBR-3; T47D; MDA-MB-468; MCF-7R; MDA-MB-231R

In vivo animal model:

-

circRNA-disease information
Expression pattern:
DN
Associated gene: TRAF4, Eg5
Associated microRNA: -
Biological function: Suppresses breast cancer cell viability, promotes monastrol-induced cytotoxicity, and reverses monastrol resistance.
Molecular mechanism: circRNA-MTO1 interacts with TRAF4 and sequesters TRAF4 from activating Eg5 translation, thereby inhibiting Eg5 protein level at the post-transcriptional level.
Biological pathway or process:

proliferation (inhibits); chemoresistance (inhibits); drug resistance (inhibits); other pathway/process (inhibits)

Detected method:
Q
M
Validation methods:

Microarray; RT-qPCR; Transfection; CCK8; IF (Immunofluorescence); Western Blot; RNA Pull-Down; circRIP; FISH / smFISH; Nuclear-Cytoplasmic Fractionation

Clinical significance:

-

Description:

circRNA-MTO1 is down-regulated in monastrol-resistant breast cancer cells. Its overexpression suppresses breast cancer cell viability, enhances monastrol cytotoxicity, and reverses monastrol resistance by binding TRAF4 and inhibiting TRAF4-mediated Eg5 protein translation.

Confidence score:

0.6503

Other information
Title:

Circular RNA‑MTO1 suppresses breast cancer cell viability and reverses monastrol resistance through regulating the TRAF4/Eg5 axis.

Journal: International journal of oncology
Published: 2018
PubMed ID: 30015883
Study type:

biological research

Data availability: The analyzed data sets generated during the study are available from the corresponding author on reasonable request.
Code availability: -