circRNA basic information
circBase ID: hsa_circ_0120106
Name: hsa_circ_PLEKHH2
Synonym: circPLEKHH2
Host Gene: PLEKHH2
Genomic location(hg19): -
Genomic location(hg38): -
Subcellular localization: nucleus and cytoplasm
 
 
 
 
 
 
 
Disease basic information
MONDO ID:
0005061
MONDO name: lung adenocarcinoma
Disease details: lung adenocarcinoma
Disease DO ID:
3910
Disease MeSH ID:
C538231
Disease NCIt ID:
C3512
Disease ICD11 ID:
-
Disease OMIM ID:
-
Species: Human
Species details: Homo sapiens
Tissue specimen:

LUAD tissues; adjacent non-tumor tissues; adjacent normal tissues; subcutaneous tumor tissue

Cell lines:

HBE; A549; H1650; H1299; H838; PC-9; H2228

In vivo animal model:

cell line-derived xenograft

circRNA-disease information
Expression pattern:
DN
Associated gene: IGF2BP3
Associated microRNA: -
Biological function: circPLEKHH2 acts as a tumor suppressor in LUAD by promoting autophagy and inhibiting LUAD cell proliferation, invasion, migration, and tumor growth.
Molecular mechanism: IGF2BP3 directly binds to circPLEKHH2 and inhibits its expression; circPLEKHH2 promotes autophagy and suppresses LUAD progression through inhibition of the AKT/mTOR signaling pathway.
Biological pathway or process:

autophagy (promotes); PI3K/AKT/mTOR (inhibits); proliferation (inhibits); migration (inhibits); invasion (inhibits); other pathway/process (inhibits)

Detected method:
Q
S
Validation methods:

Back-Splice Junction PCR / divergent primers PCR; RNase R Treatment; Sanger Sequencing; RT-qPCR; RNA-seq; Clinical Sample Validation; Nuclear-Cytoplasmic Fractionation; Transfection; CCK8; Colony Formation Assay; Transwell Assay; Western Blot; RIP (RNA Immunoprecipitation); IHC (Immunohistochemistry); In Vivo Animal Model; ROC Analysis; Bioinformatics Analysis

Clinical significance:

circPLEKHH2 expression in patient tissue has potential diagnostic value to distinguish LUAD tissue from normal lung tissue; ROC AUC was 0.949.

Description:

circPLEKHH2 is significantly downregulated in LUAD and functions as a tumor suppressor. It promotes autophagy and suppresses LUAD cell proliferation, migration, invasion, and xenograft tumor growth through inhibition of the AKT/mTOR signaling pathway. IGF2BP3 directly binds circPLEKHH2 and inhibits its expression, which may contribute to circPLEKHH2 downregulation in LUAD.

Confidence score:

0.8554

Other information
Title:

IGF2BP3-regulated circPLEKHH2 promotes autophagy via AKT/mTOR signaling pathway to suppress lung adenocarcinoma progression.

Journal: International journal of biological macromolecules
Published: 2025
PubMed ID: 40398791
Study type:

combined biological and clinical study

Data availability: Data will be made available on request; Supplementary data to this article can be found online at https://doi. org/10.1016/j.ijbiomac.2025.144262.
Code availability: -