circRNA basic information
circBase ID: -
Name: hsa_circ_OMA1
Synonym: -
Host Gene: OMA1
Genomic location(hg19): -
Genomic location(hg38): -
Subcellular localization: not tested
 
 
 
 
 
 
 
Disease basic information
MONDO ID:
0005265
MONDO name: inflammatory bowel disease
Disease details: Inflammatory bowel disease
Disease DO ID:
0050589
Disease MeSH ID:
D015212
Disease NCIt ID:
C3138
Disease ICD11 ID:
-
Disease OMIM ID:
-
Species: Human
Species details: Homo sapiens
Tissue specimen:

serum

Cell lines:

HT-29; 293 T-cells

In vivo animal model:

-

circRNA-disease information
Expression pattern:
DN
Associated gene: RAF1
Associated microRNA: miR-654-3p
Biological function: circRNA OMA1 overexpression alleviates DSS-induced colonic epithelial cell injury by enhancing cell viability, reducing apoptosis, decreasing cleaved caspase-3, and suppressing inflammatory cytokine secretion.
Molecular mechanism: circRNA OMA1 acts as a sponge for miR-654-3p and alleviates DSS-induced apoptosis and inflammation by modulating the miR-654-3p/RAF1 axis.
Biological pathway or process:

apoptosis (inhibits); inflammation (inhibits); ceRNA regulation (other)

Detected method:
Q
Validation methods:

RT-qPCR; Clinical Sample Validation; Bioinformatics Analysis; Luciferase Reporter Assay; Transfection; MTT; Annexin V/PI Flow Cytometry; Western Blot; ELISA

Clinical significance:

circRNA OMA1 could be a promising biomarker for the diagnosis and treatment of IBD.

Description:

circRNA OMA1 is downregulated in the serum of children with IBD and in DSS-treated HT-29 cells. Its overexpression protects against DSS-induced colonic epithelial injury by sponging miR-654-3p, thereby modulating RAF1 expression, reducing apoptosis and inflammatory cytokine release, and improving cell viability. The study suggests circRNA OMA1 as a potential biomarker and therapeutic target for pediatric IBD.

Confidence score:

0.6521

Other information
Title:

Mechanism underlying the role of the circRNA OMA1/miR-654-3p/RAF1 axis in children with inflammatory bowel disease.

Journal: Cytotechnology
Published: 2025
PubMed ID: 39867828
Study type:

combined biological and clinical study

Data availability: The datasets used and/or analyzed during the current study are available from the corresponding author on reasonable request; Supplementary material available at https://doi.org/10.1007/s10616-025-00703-z
Code availability: -