circRNA basic information
circBase ID: -
Name: hsa_circ_CDR1
Synonym: ciRS-7 / CDR1as
Host Gene: CDR1
Genomic location(hg19): -
Genomic location(hg38): -
Subcellular localization: not tested
 
 
 
 
 
 
 
Disease basic information
MONDO ID:
0005580
MONDO name: esophageal squamous cell carcinoma
Disease details: esophageal squamous cell carcinoma / ESCC
Disease DO ID:
3748
Disease MeSH ID:
C562729; D000077277
Disease NCIt ID:
C4024
Disease ICD11 ID:
1417891145
Disease OMIM ID:
-
Species: Human
Species details: Homo sapiens
Tissue specimen:

-

Cell lines:

TE1; Eca109; KYSE170

In vivo animal model:

-

circRNA-disease information
Expression pattern:
UP
Associated gene: EGFR, Akt-mTOR pathway, AGO2
Associated microRNA: miR-1299
Biological function: ciRS-7 inhibits starvation- or rapamycin-induced autophagy of ESCC cells.
Molecular mechanism: ciRS-7 functions as a miR-1299 sponge, relieves miR-1299-mediated repression of EGFR, and modulates EGFR/Akt/mTOR signaling to inhibit autophagy in ESCC cells.
Biological pathway or process:

autophagy (inhibits); EGFR (promotes); ceRNA regulation (other)

Detected method:
Q
Validation methods:

RT-qPCR; Bioinformatics Analysis; Transfection; RIP (RNA Immunoprecipitation); Luciferase Reporter Assay; Western Blot

Clinical significance:

ciRS-7 expression is abnormally increased in ESCC tissues and relates to poor clinicopathologic features of patients with ESCC.

Description:

This study identifies ciRS-7/CDR1as as a functional circRNA in ESCC cells that suppresses starvation- or rapamycin-induced autophagy. Mechanistically, ciRS-7 sponges miR-1299, rescues EGFR expression and downstream Akt/mTOR phosphorylation, and thereby counteracts miR-1299-induced autophagy.

Confidence score:

0.5802

Other information
Title:

Circular RNA ciRS-7 inhibits autophagy of ESCC cells by functioning as miR-1299 sponge to target EGFR signaling.

Journal: Journal of cellular biochemistry
Published: 2020
PubMed ID: 31490018
Study type:

biological research

Data availability: -
Code availability: -