circRNA basic information
circBase ID: hsa_circ_0002891
Name: hsa_circ_PDIA3
Synonym: circPDIA3
Host Gene: PDIA3
Genomic location(hg19): chr15:44048846-44055404:+
Genomic location(hg38): chr15:43756648-43763206:+
Subcellular localization: cytoplasm
 
 
 
 
 
 
 
Disease basic information
MONDO ID:
0005575
MONDO name: colorectal cancer
Disease details: colorectal cancer
Disease DO ID:
5672, 9256
Disease MeSH ID:
-
Disease NCIt ID:
C4978
Disease ICD11 ID:
-
Disease OMIM ID:
114500
Species: Human
Species details: Homo sapiens
Tissue specimen:

CRC tissues; corresponding adjacent normal tissues; OXA-sensitive tumor tissues; OXA-resistant tumor tissues; tumor tissues

Cell lines:

HCT116; HT29; DLD1; SW480; SW620; Caco-2; NCM460; HEK-293 T; HCT116-OXR; HT29-OXR; RKO

In vivo animal model:

patient-derived xenograft; cell line-derived xenograft

circRNA-disease information
Expression pattern:
UP
Associated gene: GSDME, GSDME-C domain, GSDME-N domain, ZDHHC3, ZDHHC17, XBP1, AGO2, PDIA3
Associated microRNA: miR-449a
Biological function: circPDIA3 promotes oxaliplatin chemoresistance in colorectal cancer by inhibiting GSDME-dependent pyroptosis; high circPDIA3 expression is associated with recurrence/metastasis and poor disease-free survival after OXA-based chemotherapy.
Molecular mechanism: circPDIA3 directly binds the GSDME-C domain and enhances its autoinhibitory interaction with GSDME-N by inhibiting ZDHHC3/ZDHHC17-dependent GSDME-C palmitoylation; circPDIA3 also sponges miR-449a to upregulate XBP1, while XBP1 promotes circPDIA3 expression, forming a positive feedback loop.
Biological pathway or process:

pyroptosis (inhibits); chemoresistance (promotes); drug resistance (promotes); ceRNA regulation (promotes); other pathway/process (inhibits); apoptosis (inhibits); proliferation (promotes)

Detected method:
Q
B
S
M
Validation methods:

Back-Splice Junction PCR / divergent primers PCR; RNase R Treatment; Sanger Sequencing; Actinomycin D / DRB Stability Assay; RT-qPCR; RNA-seq; Microarray; Northern Blot; FISH / smFISH; IF (Immunofluorescence); Nuclear-Cytoplasmic Fractionation; Clinical Sample Validation; RIP (RNA Immunoprecipitation); RNA Pull-Down; Co-IP; ChIP / ChIP-seq; Luciferase Reporter Assay; Transfection; CCK8; EdU Staining; Colony Formation Assay; Annexin V/PI Flow Cytometry; In Vivo Animal Model; H&E Staining; IHC (Immunohistochemistry); ELISA; Western Blot; Cohort Study; Survival Analysis; Bioinformatics Analysis

Clinical significance:

circPDIA3 is closely associated with unfavorable clinical prognosis and can act as an independent predictor of OXA response in CRC patients; high circPDIA3 expression is associated with recurrence/metastasis and lower DFS.

Description:

circPDIA3 is an upregulated PDIA3-derived circular RNA in CRC, especially in oxaliplatin-resistant tumors, and high expression predicts poorer disease-free survival after OXA-based therapy. Functionally, circPDIA3 promotes chemoresistance by suppressing GSDME-dependent pyroptosis through direct binding to the GSDME-C domain and inhibition of ZDHHC3/ZDHHC17-mediated palmitoylation. It also forms a circPDIA3/miR-449a/XBP1 positive feedback loop that further increases circPDIA3 expression and reinforces OXA resistance.

Confidence score:

0.9043

Other information
Title:

CircPDIA3/miR-449a/XBP1 feedback loop curbs pyroptosis by inhibiting palmitoylation of the GSDME-C domain to induce chemoresistance of colorectal cancer.

Journal: Drug resistance updates : reviews and commentaries in antimicrobial and anticancer chemotherapy
Published: 2024
PubMed ID: 38861804
Study type:

combined biological and clinical study

Data availability: GSE120985; GSE126094; GSE138589; Supplementary Table S1; Supplementary data at doi:10.1016/j.drup.2024.101097; datasets available from corresponding author Yong Li upon reasonable request
Code availability: -