circRNA basic information
circBase ID: hsa_circ_0116061
Name: hsa_circ_BRWD1
Synonym: circ-BRWD1
Host Gene: BRWD1
Genomic location(hg19): -
Genomic location(hg38): -
Subcellular localization: cytoplasm
 
 
 
 
 
 
 
Disease basic information
MONDO ID:
0005178
MONDO name: osteoarthritis
Disease details: osteoarthritis
Disease DO ID:
8398
Disease MeSH ID:
D010003
Disease NCIt ID:
C3293
Disease ICD11 ID:
558562409
Disease OMIM ID:
-
Species: Human
Species details: Homo sapiens
Tissue specimen:

cartilage tissue; exosomes

Cell lines:

CHON-001

In vivo animal model:

-

circRNA-disease information
Expression pattern:
UP
Associated gene: TRAF6, Ago2
Associated microRNA: miR-1277
Biological function: promotes IL-1beta-induced chondrocyte injury by inhibiting cell viability and facilitating apoptosis, inflammation and ECM degradation
Molecular mechanism: acts as a miRNA sponge for miR-1277 to positively regulate TRAF6 expression
Biological pathway or process:

apoptosis (promotes); proliferation (inhibits); inflammation (promotes); fibrosis (not specified)

Detected method:
Q
Validation methods:

RT-qPCR; RNase R Treatment; Nuclear-Cytoplasmic Fractionation; Clinical Sample Validation; Luciferase Reporter Assay; RIP (RNA Immunoprecipitation); Transfection; CCK8; EdU Staining; Annexin V/PI Flow Cytometry; ELISA; Western Blot

Clinical significance:

-

Description:

circ-BRWD1 is upregulated in osteoarthritis cartilage and IL-1beta-stimulated chondrocytes and is enriched in the cytoplasm. It is transmitted via exosomes and aggravates IL-1beta-induced chondrocyte injury by sponging miR-1277 to increase TRAF6, thereby promoting apoptosis, inflammation and ECM degradation while reducing cell viability/proliferation.

Confidence score:

0.7631

Other information
Title:

Exosomal circ-BRWD1 contributes to osteoarthritis development through the modulation of miR-1277/TRAF6 axis.

Journal: Arthritis research & therapy
Published: 2021
PubMed ID: 34082824
Study type:

combined biological and clinical study

Data availability: The analyzed data sets generated during the present study are available from the corresponding author on reasonable request.
Code availability: -