circRNA basic information
circBase ID: -
Name: hsa_circ_PSMC3
Synonym: CircPSMC3
Host Gene: PSMC3
Genomic location(hg19): -
Genomic location(hg38): -
Subcellular localization: not tested
 
 
 
 
 
 
 
Disease basic information
MONDO ID:
0005233
MONDO name: non-small cell lung carcinoma
Disease details: non-small cell lung cancer
Disease DO ID:
3908
Disease MeSH ID:
D002289
Disease NCIt ID:
C2926
Disease ICD11 ID:
-
Disease OMIM ID:
-
Species: Human
Species details: Homo sapiens
Tissue specimen:

NSCLC tissues; corresponding normal tissues / adjacent normal tissues

Cell lines:

H1299; H157; GLC-82; A549; H358

In vivo animal model:

-

circRNA-disease information
Expression pattern:
DN
Associated gene: NME2
Associated microRNA: miR-182-5p
Biological function: inhibits invasion and migration (metastasis-related behaviors) of NSCLC cells
Molecular mechanism: circPSMC3 sponges miR-182-5p to upregulate NME2 (ceRNA mechanism)
Biological pathway or process:

migration (inhibits); invasion (inhibits); metastasis (inhibits); ceRNA regulation (other)

Detected method:
Q
Validation methods:

RT-qPCR; Clinical Sample Validation; Pearson correlation analysis; Transfection; Transwell Assay; Wound Healing Assay; Luciferase Reporter Assay; Western Blot

Clinical significance:

Lower circPSMC3 expression was closely associated with lymph node metastasis and higher TNM stage in NSCLC patients.

Description:

CircPSMC3 is down-regulated in NSCLC tissues and its low expression is associated with lymph node metastasis and higher TNM stage. Functionally, circPSMC3 suppresses NSCLC cell migration and invasion by sponging miR-182-5p, thereby upregulating NME2.

Confidence score:

0.686

Other information
Title:

CircPSMC3 Suppresses Migration and Invasion of Non-Small Cell Lung Cancer Cells via miR-182-5p/NME2 Axis.

Journal: Medical science monitor : international medical journal of experimental and clinical research
Published: 2020
PubMed ID: 32386284
Study type:

combined biological and clinical study

Data availability: -
Code availability: -