circRNA basic information
circBase ID: hsa_circ_0000284
Name: hsa_circ_HIPK3
Synonym: circHIPK3
Host Gene: HIPK3
Genomic location(hg19): chr11:33307958-33309057:+
Genomic location(hg38): chr11:33286412-33287511:+
Subcellular localization: not tested
 
 
 
 
 
 
 
Disease basic information
MONDO ID:
0007256
MONDO name: hepatocellular carcinoma
Disease details: hepatocellular carcinoma
Disease DO ID:
684, 686
Disease MeSH ID:
D006528
Disease NCIt ID:
C3099
Disease ICD11 ID:
1294035808
Disease OMIM ID:
114550
Species: Human
Species details: Homo sapiens
Tissue specimen:

hepatocellular carcinoma tissues; adjacent noncancerous tissues

Cell lines:

SMMC-7721; Huh7; HepG2; Hep3B; HEK293T

In vivo animal model:

-

circRNA-disease information
Expression pattern:
UP
Associated gene: MRP4
Associated microRNA: miR-124-3p, miR-4524-5p
Biological function: Promotes MRP4 upregulation and may contribute to MRP4-related proliferation and multiple drug resistance in HCC.
Molecular mechanism: circHIPK3 acts as a competitive endogenous RNA (ceRNA) to sponge miR-124-3p and miR-4524-5p, thereby reducing free miRNAs and increasing MRP4 protein expression.
Biological pathway or process:

ceRNA regulation (promotes); proliferation (other); drug resistance (other)

Detected method:
Q
Validation methods:

RT-qPCR; Luciferase Reporter Assay; circRIP; Transfection; Western Blot; Clinical Sample Validation

Clinical significance:

-

Description:

circHIPK3 is up-regulated in HCC tissues and functions as a ceRNA that sponges miR-124-3p and miR-4524-5p. By reducing the availability of these miRNAs, circHIPK3 increases MRP4 protein expression, which may relate to proliferation and multidrug resistance in HCC.

Confidence score:

0.6427

Other information
Title:

Regulation of MRP4 Expression by circHIPK3 via Sponging miR-124-3p/miR-4524-5p in Hepatocellular Carcinoma.

Journal: Biomedicines
Published: 2021
PubMed ID: 33946595
Study type:

combined biological and clinical study

Data availability: The data presented in this study are available on request from the corresponding authors.
Code availability: -