circRNA basic information
circBase ID: -
Name: -
Synonym: circRNA_103762
Host Gene: -
Genomic location(hg19): -
Genomic location(hg38): -
Subcellular localization: not tested
 
 
 
 
 
 
 
Disease basic information
MONDO ID:
0005233
MONDO name: non-small cell lung carcinoma
Disease details: non-small cell lung cancer
Disease DO ID:
3908
Disease MeSH ID:
D002289
Disease NCIt ID:
C2926
Disease ICD11 ID:
-
Disease OMIM ID:
-
Species: Human
Species details: Homo sapiens
Tissue specimen:

NSCLC tissues; adjacent normal tissues; normal lung samples

Cell lines:

Beas-2B; A549; NCI-H1299; H1650; H358; H358/CDDP

In vivo animal model:

-

circRNA-disease information
Expression pattern:
UP
Associated gene: DNA damage inducible transcript 3 (CHOP)
Associated microRNA: -
Biological function: Promotes proliferation, migration, invasion and enhances multidrug resistance in NSCLC; inhibits CHOP expression.
Molecular mechanism: Enhances MDR by inhibiting CHOP expression in NSCLC cells.
Biological pathway or process:

proliferation (promotes); migration (promotes); invasion (promotes); drug resistance (promotes)

Detected method:
Q
M
Validation methods:

Microarray; Bioinformatics Analysis; RT-qPCR; Clinical Sample Validation; Survival Analysis; Transfection; CCK8; Transwell Assay; Western Blot

Clinical significance:

Higher circRNA_103762 expression in NSCLC patients was associated with lower survival rate.

Description:

circRNA_103762 is up-regulated in NSCLC tissues and cell lines and is associated with worse overall survival. Functionally, its knockdown suppresses proliferation/migration/invasion and reduces multidrug resistance, at least partly by relieving inhibition of CHOP expression.

Confidence score:

0.6719

Other information
Title:

CircRNA_103762 promotes multidrug resistance in NSCLC by targeting DNA damage inducible transcript 3 (CHOP).

Journal: Journal of clinical laboratory analysis
Published: 2020
PubMed ID: 32118311
Study type:

combined biological and clinical study

Data availability: GSE112214
Code availability: -