circRNA basic information
circBase ID: hsa_circ_0002940
Name: hsa_circ_CCT2
Synonym: cCCT2 / SenExo-cCCT2
Host Gene: CCT2
Genomic location(hg19): chr12:69983264-69987393:+
Genomic location(hg38): chr12:69589484-69593613:+
Subcellular localization: nucleus and cytoplasm
 
 
 
 
 
 
 
Disease basic information
MONDO ID:
0005184
MONDO name: pancreatic ductal adenocarcinoma
Disease details: pancreatic ductal adenocarcinoma / PDAC
Disease DO ID:
3498, 3587
Disease MeSH ID:
D021441
Disease NCIt ID:
C9120
Disease ICD11 ID:
581089833
Disease OMIM ID:
-
Species: Human
Species details: Homo sapiens
Tissue specimen:

pancreatic cancer tissue; tumor tissue

Cell lines:

PANC-1; BxPC-3; Capan-1; MIA PaCa-2; AsPC-1; SW1990; 293T/293F

In vivo animal model:

cell line-derived xenograft; patient-derived xenograft

circRNA-disease information
Expression pattern:
UP
Associated gene: SLX4, IPO13, UBC9, CXCL10, PD-L1
Associated microRNA: -
Biological function: Promotes chemotherapy-induced senescence, increases chemosensitivity, promotes CD8+ T-cell infiltration via SASP (CXCL10), but induces immune escape by upregulating PD-L1.
Molecular mechanism: cCCT2 competitively binds IPO13, blocks UBC9 nuclear import, reduces SLX4 SUMOylation, increases SLX4 export and ubiquitin-proteasome degradation, inhibits SLX4 condensate-mediated DNA damage repair, leading to DNA damage accumulation and senescence; SASP CXCL10 recruits CD8+ T-cells; PD-L1 upregulation causes exhaustion and is reversible by anti-PD-L1.
Biological pathway or process:

m6A modification (not specified)

Detected method:
Q
H
S
Validation methods:

circRNA-seq; RT-qPCR; ISH (In Situ Hybridization); Back-Splice Junction PCR / divergent primers PCR; RNase R Treatment; Sanger Sequencing; Actinomycin D / DRB Stability Assay; FISH / smFISH; Nuclear-Cytoplasmic Fractionation; Clinical Sample Validation; Transfection; CCK8; EdU Staining; Annexin V/PI Flow Cytometry; Western Blot; RNA Pull-Down; RIP (RNA Immunoprecipitation); CLIP; IF (Immunofluorescence); Luciferase Reporter Assay; Transwell Assay; ELISA; In Vivo Animal Model; IHC (Immunohistochemistry); H&E Staining; Survival Analysis; Bioinformatics Analysis

Clinical significance:

High cCCT2 expression was associated with better prognosis; cCCT2 was upregulated in chemotherapy-responsive PDAC tissues and PDOs and suggested as a predictive biomarker for PDAC treatment.

Description:

In PDAC, cCCT2 (hsa_circ_0002940) is upregulated in chemotherapy-responsive cases and promotes FOLFIRINOX-induced senescence and chemosensitivity by impairing SLX4-dependent DNA damage repair via IPO13/UBC9-regulated SUMOylation. Senescent tumor cells secrete CXCL10 to recruit CD8+ T-cells but also upregulate PD-L1, enabling immune escape; sequential anti-PD-L1 therapy restores cytotoxic function. SenExo-cCCT2 is an exosome-based delivery strategy to exploit these effects therapeutically.

Confidence score:

0.8988

Other information
Title:

SenExo-cCCT2 Reprograms Senescence Response and Anti-Tumor Immunity Following FOLFIRINOX Chemotherapy in Pancreatic Ductal Adenocarcinoma.

Journal: Advanced science (Weinheim, Baden-Wurttemberg, Germany)
Published: 2025
PubMed ID: 40686389
Study type:

combined biological and clinical study

Data availability: The data that support the findings of this study are available from the corresponding author upon reasonable request.
Code availability: -