circRNA basic information
circBase ID: hsa_circ_0006101
Name: hsa_circ_ORC2
Synonym: circ_ORC2
Host Gene: -
Genomic location(hg19): chr2:201784944-201802689:-
Genomic location(hg38): chr2:200920221-200937966:-
Subcellular localization: cytoplasm
 
 
 
 
 
 
 
Disease basic information
MONDO ID:
0009807
MONDO name: osteosarcoma
Disease details: osteosarcoma / OS
Disease DO ID:
3347
Disease MeSH ID:
D012516
Disease NCIt ID:
C9145
Disease ICD11 ID:
-
Disease OMIM ID:
-
Species: Human
Species details: Homo sapiens
Tissue specimen:

-

Cell lines:

143B; HOS; MG-63; Saos-2; U-2 OS; hFOB 1.19

In vivo animal model:

-

circRNA-disease information
Expression pattern:
UP
Associated gene: Ago2 protein, PTEN, Akt
Associated microRNA: miR-19a
Biological function: promotes osteosarcoma cell growth, proliferation and invasion; inhibits apoptosis
Molecular mechanism: circ_ORC2 binds miR-19a, enhances miR-19a expression/function, inhibits PTEN expression and activates Akt pathway
Biological pathway or process:

PI3K/AKT (promotes); proliferation (promotes); invasion (promotes); apoptosis (inhibits); ceRNA regulation (other)

Detected method:
Q
Validation methods:

RT-qPCR; Nuclear-Cytoplasmic Fractionation; RIP (RNA Immunoprecipitation); RNA Pull-Down; Luciferase Reporter Assay; Transfection; Western Blot; BrdU; Annexin V/PI Flow Cytometry; Transwell Assay; Bioinformatics Analysis

Clinical significance:

-

Description:

hsa_circ_0006101/circ_ORC2 is up-regulated in human osteosarcoma cell lines and is mainly cytoplasmic. It binds miR-19a and enhances miR-19a-mediated repression of PTEN, leading to Akt pathway activation, increased proliferation and invasion, and reduced apoptosis in osteosarcoma cells.

Confidence score:

0.6853

Other information
Title:

Circ_ORC2 enhances the regulatory effect of miR-19a on its target gene PTEN to affect osteosarcoma cell growth.

Journal: Biochemical and biophysical research communications
Published: 2019
PubMed ID: 31103262
Study type:

biological research

Data availability: -
Code availability: -