circRNA basic information
circBase ID: -
Name: hsa_circ_ATXN7
Synonym: -
Host Gene: ATXN7
Genomic location(hg19): -
Genomic location(hg38): -
Subcellular localization: not tested
 
 
 
 
 
 
 
Disease basic information
MONDO ID:
0005233
MONDO name: non-small cell lung carcinoma
Disease details: non-small cell lung cancer
Disease DO ID:
3908
Disease MeSH ID:
D002289
Disease NCIt ID:
C2926
Disease ICD11 ID:
-
Disease OMIM ID:
-
Species: Human
Species details: Homo sapiens
Tissue specimen:

NSCLC tissues; adjacent normal tissues / adjacent non-tumor tissues

Cell lines:

A549; SPC-A1

In vivo animal model:

-

circRNA-disease information
Expression pattern:
UP
Associated gene: -
Associated microRNA: -
Biological function: Promotes proliferation and invasion of NSCLC cells (oncogenic role).
Molecular mechanism: Mechanism not experimentally determined; authors suggest circATXN7 may act as a miRNA sponge.
Biological pathway or process:

proliferation (promotes); invasion (promotes)

Detected method:
Q
Validation methods:

RT-qPCR; Sanger Sequencing; Back-Splice Junction PCR / divergent primers PCR; Clinical Sample Validation; Transfection; MTT; EdU Staining; Colony Formation Assay; Transwell Assay; Survival Analysis

Clinical significance:

High circATXN7 levels were associated with shorter survival time, but not statistically significant; no significant associations with most clinicopathological characteristics.

Description:

circATXN7 is upregulated in NSCLC tumor tissues. Functional knockdown experiments indicate circATXN7 promotes NSCLC cell proliferation and invasion in vitro, supporting an oncogenic role; its miRNA-sponge mechanism is proposed but not validated in this study.

Confidence score:

0.7353

Other information
Title:

Circular RNA ATXN7 is upregulated in non-small cell lung cancer and promotes disease progression.

Journal: Oncology letters
Published: 2019
PubMed ID: 31186686
Study type:

combined biological and clinical study

Data availability: The datasets used or analyzed during the current study are available from the corresponding author on reasonable request.
Code availability: -