circRNA basic information
circBase ID: -
Name: hsa_circ_LRP6
Synonym: circLRP6 / hsa_circRNA_101014
Host Gene: LRP6
Genomic location(hg19): -
Genomic location(hg38): -
Subcellular localization: cytoplasm
 
 
 
 
 
 
 
Disease basic information
MONDO ID:
0002531
MONDO name: skin neoplasm
Disease details: arsenite-induced malignant transformation of human keratinocytes / arsenite-induced carcinogenesis / human skin cancers
Disease DO ID:
3165
Disease MeSH ID:
D012878
Disease NCIt ID:
C3372
Disease ICD11 ID:
-
Disease OMIM ID:
-
Species: Human
Species details: Homo sapiens
Tissue specimen:

-

Cell lines:

HaCaT; T-HaCaT

In vivo animal model:

-

circRNA-disease information
Expression pattern:
UP
Associated gene: ZEB1
Associated microRNA: miR-455
Biological function: circLRP6 promotes arsenite-induced EMT, malignant transformation, colony formation, migration, and invasion in T-HaCaT cells.
Molecular mechanism: circLRP6 acts as a cytoplasmic miR-455 sponge, thereby up-regulating the miR-455 target ZEB1 and inducing EMT during arsenite-induced malignant transformation.
Biological pathway or process:

EMT (promotes); migration (promotes); invasion (promotes); ceRNA regulation (promotes); other pathway/process (promotes)

Detected method:
Q
Validation methods:

Back-Splice Junction PCR / divergent primers PCR; RNase R Treatment; Sanger Sequencing; RT-qPCR; FISH / smFISH; IF (Immunofluorescence); Nuclear-Cytoplasmic Fractionation; RNA Pull-Down; Luciferase Reporter Assay; Transfection; Colony Formation Assay; Transwell Assay; Western Blot; Bioinformatics Analysis

Clinical significance:

-

Description:

circLRP6 is up-regulated during arsenite-induced malignant transformation of human HaCaT keratinocytes. It functions mainly in the cytoplasm as a miR-455 sponge, leading to increased ZEB1 expression and promotion of EMT, colony formation, migration, and invasion.

Confidence score:

0.7909

Other information
Title:

CircLRP6 Regulation of ZEB1 via miR-455 Is Involved in the Epithelial-Mesenchymal Transition During Arsenite-Induced Malignant Transformation of Human Keratinocytes.

Journal: Toxicological sciences : an official journal of the Society of Toxicology
Published: 2018
PubMed ID: 29216394
Study type:

biological research

Data availability: Supplementary data are available at Toxicological Sciences online.
Code availability: -