circRNA basic information
circBase ID: hsa_circ_0000615
Name: hsa_circ_ZNF609
Synonym: circZNF609
Host Gene: ZNF609
Genomic location(hg19): chr15:64791491-64792365:+
Genomic location(hg38): chr15:64499292-64500166:+
Subcellular localization: cytoplasm
 
 
 
 
 
 
 
Disease basic information
MONDO ID:
0012817
MONDO name: Ewing sarcoma
Disease details: Ewing’s sarcoma / EwS
Disease DO ID:
3369
Disease MeSH ID:
D012512
Disease NCIt ID:
C4817
Disease ICD11 ID:
458106328
Disease OMIM ID:
612219
Species: Human
Species details: Homo sapiens
Tissue specimen:

-

Cell lines:

A673; 6647; TC32; A4573; SKES-1; AB678; HEK 293T

In vivo animal model:

-

circRNA-disease information
Expression pattern:
UP
Associated gene: EWS::FLI1, FLI1 3′ UTR
Associated microRNA: miR-145-5p
Biological function: promotes cell proliferation and metastasis while inhibiting apoptosis
Molecular mechanism: circZNF609 acts as a molecular sponge for miR-145-5p, preventing miR-145-5p from binding to the 3′ UTR of EWS::FLI1 and relieving translational repression of EWS::FLI1.
Biological pathway or process:

proliferation (promotes); apoptosis (inhibits); metastasis (promotes); ceRNA regulation (promotes)

Detected method:
Q
Validation methods:

RT-qPCR; divergent primers PCR; RNase R Treatment; Sanger Sequencing; FISH / smFISH; Transfection; MTT; Annexin V/PI Flow Cytometry; Colony Formation Assay; Western Blot; IF (Immunofluorescence); Luciferase Reporter Assay; Bioinformatics Analysis

Clinical significance:

-

Description:

In Ewing’s sarcoma cell lines, circZNF609 is highly expressed and predominantly cytoplasmic. It promotes proliferation and tumorigenic behaviors and inhibits apoptosis by sponging miR-145-5p, thereby relieving miR-145-5p-mediated translational repression of EWS::FLI1.

Confidence score:

0.7446

Other information
Title:

Circuitous Ways of EWS::FLI1 Using Circular RNA ZNF609 to Evade Translational Repression by miR-145 in Ewing's Sarcoma.

Journal: Biomedicines
Published: 2026
PubMed ID: 41595666
Study type:

biological research

Data availability: The original contributions presented in this study are included in the article/Supplementary Material.
Code availability: -