circRNA basic information
circBase ID: -
Name: hsa_circ_SCYL2
Synonym: -
Host Gene: SCYL2
Genomic location(hg19): chr12:100676721-100691953:+
Genomic location(hg38): chr12:100282943-100298175:+
Subcellular localization: not tested
 
 
 
 
 
 
 
Disease basic information
MONDO ID:
0007254
MONDO name: breast cancer
Disease details: breast cancer
Disease DO ID:
1612
Disease MeSH ID:
-
Disease NCIt ID:
C9335
Disease ICD11 ID:
1047754165
Disease OMIM ID:
-
Species: Human
Species details: Homo sapiens
Tissue specimen:

breast cancer tissues; normal adjacent tissues

Cell lines:

MCF-7; MDA-MB-231

In vivo animal model:

-

circRNA-disease information
Expression pattern:
DN
Associated gene: OSR1, TGF-beta signaling pathway
Associated microRNA: -
Biological function: Inhibits breast cancer cell migration and invasion; suppresses EMT progression.
Molecular mechanism: May regulate EMT via OSR1/TGF-beta axis based on circRNA-miRNA-mRNA network predictions.
Biological pathway or process:

migration (inhibits); invasion (inhibits); EMT (inhibits); ceRNA regulation (other); TGF-beta/SMAD (other)

Detected method:
Q
S
Validation methods:

RNA-seq; RT-qPCR; Back-Splice Junction PCR / divergent primers PCR; Sanger Sequencing; Clinical Sample Validation; Transfection; Transwell Assay

Clinical significance:

May serve as biomarkers during tumor metastasis.

Description:

circSCYL2 (from SCYL2 exon 2-4) is down-regulated in breast cancer tissues/cell lines and its overexpression suppresses EMT-associated migration and invasion in MCF-7 and MDA-MB-231 cells. Mechanistically, the study’s network analysis suggests circSCYL2 may influence EMT via an OSR1/TGF-beta signaling axis, but the interactions were stated to require further experimental validation.

Confidence score:

0.673

Other information
Title:

EMT related circular RNA expression profiles identify circSCYL2 as a novel molecule in breast tumor metastasis.

Journal: International journal of molecular medicine
Published: 2020
PubMed ID: 32236616
Study type:

combined biological and clinical study

Data availability: available from the corresponding author on reasonable request
Code availability: -