circRNA basic information
circBase ID: -
Name: hsa_circ_RNF20
Synonym: circRNA ring finger protein 20 / circRNF20
Host Gene: RNF20
Genomic location(hg19): -
Genomic location(hg38): -
Subcellular localization: cytoplasm
 
 
 
 
 
 
 
Disease basic information
MONDO ID:
0007254
MONDO name: breast cancer
Disease details: breast cancer / BC
Disease DO ID:
1612
Disease MeSH ID:
-
Disease NCIt ID:
C9335
Disease ICD11 ID:
1047754165
Disease OMIM ID:
-
Species: Human
Species details: Homo sapiens
Tissue specimen:

tumor tissues

Cell lines:

MDA‐MB‐157; MDA‐MB‐468; MDA‐MB‐231; MDA‐MB‐453; MCF‐10A

In vivo animal model:

cell line-derived xenograft

circRNA-disease information
Expression pattern:
UP
Associated gene: IGF2BP2, HuR, CDCA4
Associated microRNA: -
Biological function: facilitates breast cancer cell proliferation
Molecular mechanism: IGF2BP2 binds circRNF20 to prevent its degradation and promote circRNF20 binding to HuR, which increases CDCA4 expression.
Biological pathway or process:

proliferation (promotes); other pathway/process (promotes)

Detected method:
Q
Validation methods:

RT-qPCR; RIP (RNA Immunoprecipitation); RNA Pull-Down; Actinomycin D / DRB Stability Assay; FISH / smFISH; IF (Immunofluorescence); Transfection; CCK8; Colony Formation Assay; EdU Staining; Western Blot; In Vivo Animal Model; IHC (Immunohistochemistry); Bioinformatics Analysis

Clinical significance:

-

Description:

circRNF20 is up-regulated in breast cancer cell lines and promotes cancer cell proliferation. Mechanistically, IGF2BP2 stabilizes circRNF20, enabling circRNF20 to bind HuR and enhance CDCA4 expression, forming an IGF2BP2-circRNF20-HuR-CDCA4 pro-proliferative axis.

Confidence score:

0.7799

Other information
Title:

IGF2BP2-induced circRNF20 facilitates breast cancer cell proliferation via the HuR/CDCA4 axis.

Journal: The Kaohsiung journal of medical sciences
Published: 2025
PubMed ID: 39969065
Study type:

biological research

Data availability: The data that support the findings of this study are available from the corresponding author upon reasonable request.
Code availability: -