circRNA basic information
circBase ID: hsa_circ_0000479
Name: hsa_circ_EPSTI1
Synonym: circEPSTI1 / circ0000479
Host Gene: EPSTI1
Genomic location(hg19): -
Genomic location(hg38): -
Subcellular localization: cytoplasm
 
 
 
 
 
 
 
Disease basic information
MONDO ID:
0007254
MONDO name: breast cancer
Disease details: HER2-positive breast cancer
Disease DO ID:
1612
Disease MeSH ID:
-
Disease NCIt ID:
C9335
Disease ICD11 ID:
1047754165
Disease OMIM ID:
-
Species: Human
Species details: Homo sapiens
Tissue specimen:

HER2-positive breast cancer tissues; adjacent normal tissues

Cell lines:

SKBR3; BT474; MCF-10A

In vivo animal model:

-

circRNA-disease information
Expression pattern:
UP
Associated gene: ERBB3, Ago2
Associated microRNA: miR-145
Biological function: promotes proliferation, migration, and invasion of HER2-positive breast cancer cells
Molecular mechanism: ceRNA mechanism: circEPSTI1 sponges miR-145 to regulate ERBB3
Biological pathway or process:

proliferation (promotes); migration (promotes); invasion (promotes); ceRNA regulation (promotes)

Detected method:
Q
Validation methods:

RT-qPCR; RNase R Treatment; Nuclear-Cytoplasmic Fractionation; Transfection; CCK8; Wound Healing Assay; Transwell Assay; RIP (RNA Immunoprecipitation); Luciferase Reporter Assay; Western Blot; Clinical Sample Validation; Bioinformatics Analysis

Clinical significance:

circEPSTI1 could be used as a novel biomarker and potential therapeutic target for breast cancer.

Description:

circEPSTI1 is up-regulated in HER2-positive breast cancer tissues and cell lines and mainly localizes to the cytoplasm. It promotes proliferation, migration and invasion by acting as a ceRNA sponge for miR-145, thereby relieving repression of ERBB3.

Confidence score:

0.7785

Other information
Title:

CircEPSTI1 Promotes the Proliferation of HER2-Positive Breast Cancer Cells via circEPSTI1/miR-145/ERBB3 Axis.

Journal: Journal of oncology
Published: 2022
PubMed ID: 36059813
Study type:

combined biological and clinical study

Data availability: The data are available from the corresponding author Xing Li upon request via email (lixing@sysucc.org.cn).
Code availability: -