circRNA basic information
circBase ID: -
Name: hsa_circ_RAPGEF5
Synonym: circRAPGEF5
Host Gene: RAPGEF5
Genomic location(hg19): -
Genomic location(hg38): -
Subcellular localization: cytoplasm
 
 
 
 
 
 
 
Disease basic information
MONDO ID:
0005061
MONDO name: lung adenocarcinoma
Disease details: lung adenocarcinoma / LUAD
Disease DO ID:
3910
Disease MeSH ID:
C538231
Disease NCIt ID:
C3512
Disease ICD11 ID:
-
Disease OMIM ID:
-
Species: Human
Species details: Homo sapiens
Tissue specimen:

LUAD tissue; precancerous tissues; tumor tissues of nude mice

Cell lines:

A549; H1299; HCC827; BEAS-2B

In vivo animal model:

cell line-derived xenograft; genetically engineered animal model

circRNA-disease information
Expression pattern:
UP
Associated gene: IGF2BP2, NUP160, ZEB1, METTL3
Associated microRNA: miRNA-126-3p
Biological function: Suppresses autophagy and promotes malignant phenotypes including proliferation, migration, invasion, colony formation, EMT, tumor growth and metastasis in LUAD.
Molecular mechanism: m6A-modified circRAPGEF5 binds IGF2BP2 (m6A reader), facilitating IGF2BP2-mediated stabilization of NUP160 mRNA, leading to autophagy suppression and LUAD progression/metastasis.
Biological pathway or process:

autophagy (inhibits); proliferation (promotes); migration (promotes); invasion (promotes); EMT (promotes); metastasis (promotes); ceRNA regulation (other)

Detected method:
Q
Validation methods:

RT-qPCR; Clinical Sample Validation; FISH / smFISH; RNA Pull-Down; RIP (RNA Immunoprecipitation); MeRIP / MeRIP-seq; Transfection; CCK8; EdU Staining; Colony Formation Assay; Transwell Assay; Western Blot; IF (Immunofluorescence); Actinomycin D / DRB Stability Assay; In Vivo Animal Model; H&E Staining; Bioinformatics Analysis; Survival Analysis

Clinical significance:

Serum exosomal circRAPGEF5 may serve as a marker for identifying LUAD tumors; Patients with high expression of circRAPGEF5 had shorter survival.

Description:

circRAPGEF5 is up-regulated in LUAD and predominantly localizes in the cytoplasm. It binds the m6A reader IGF2BP2 and promotes IGF2BP2-dependent stabilization of NUP160 mRNA, suppressing autophagy and thereby enhancing LUAD proliferation, EMT, invasion, and metastasis, including in xenograft models.

Confidence score:

0.8326

Other information
Title:

M6A-Methylated circRAPGEF5 drives lung adenocarcinoma progression and metastasis via IGF2BP2/NUP160-mediated autophagy suppression.

Journal: Molecular cancer
Published: 2025
PubMed ID: 40629330
Study type:

combined biological and clinical study

Data availability: -
Code availability: -