circRNA basic information
circBase ID: hsa_circ_0000745
Name: hsa_circ_SPECC1
Synonym: circSPECC1
Host Gene: SPECC1
Genomic location(hg19): chr17:20107645-20109225:+
Genomic location(hg38): chr17:20204332-20205912:+
Subcellular localization: not tested
 
 
 
 
 
 
 
Disease basic information
MONDO ID:
0021042
MONDO name: glioma
Disease details: glioma
Disease DO ID:
-
Disease MeSH ID:
D005910
Disease NCIt ID:
C3059
Disease ICD11 ID:
-
Disease OMIM ID:
-
Species: Human
Species details: Homo sapiens
Tissue specimen:

-

Cell lines:

U251; U87; HEK293

In vivo animal model:

-

circRNA-disease information
Expression pattern:
UP
Associated gene: YB-1, HIP1, AKT
Associated microRNA: miR-615-5p
Biological function: Promotes proliferation, migration and invasion and inhibits apoptosis in glioma cells.
Molecular mechanism: YB-1-regulated circSPECC1 acts as a ceRNA/miRNA sponge for miR-615-5p to upregulate HIP1 and activate HIP1/AKT signaling.
Biological pathway or process:

proliferation (promotes); migration (promotes); invasion (promotes); apoptosis (inhibits); PI3K/AKT (promotes); ceRNA regulation (promotes)

Detected method:
Q
Validation methods:

RIP (RNA Immunoprecipitation); RT-qPCR; Bioinformatics Analysis; RNA Pull-Down; Luciferase Reporter Assay; Transfection; CCK8; Wound Healing Assay; Transwell Assay; Annexin V/PI Flow Cytometry; Western Blot

Clinical significance:

circSPECC1 is associated with the prognosis of glioma patients.

Description:

In human glioma cell lines, circSPECC1 is upregulated and promotes proliferation, migration and invasion while inhibiting apoptosis. Mechanistically, it is regulated by YB-1 and functions as a ceRNA to sponge miR-615-5p, thereby increasing HIP1 and activating AKT signaling.

Confidence score:

0.6578

Other information
Title:

Y-box protein-1 modulates circSPECC1 to promote glioma tumorigenesis via miR-615-5p/HIP1/AKT axis.

Journal: Acta biochimica et biophysica Sinica
Published: 2023
PubMed ID: 37994157
Study type:

biological research

Data availability: -
Code availability: -