circRNA basic information
circBase ID: -
Name: hsa_circ_NFKB1
Synonym: circ-NFKB1
Host Gene: NFKB1
Genomic location(hg19): -
Genomic location(hg38): -
Subcellular localization: not tested
 
 
 
 
 
 
 
Disease basic information
MONDO ID:
0005178
MONDO name: osteoarthritis
Disease details: Osteoarthritis / OA
Disease DO ID:
8398
Disease MeSH ID:
D010003
Disease NCIt ID:
C3293
Disease ICD11 ID:
558562409
Disease OMIM ID:
-
Species: Human
Species details: Homo sapiens
Tissue specimen:

cartilage tissue

Cell lines:

CHON-001; 293 T

In vivo animal model:

other disease animal model

circRNA-disease information
Expression pattern:
UP
Associated gene: ERBB4, Ago2
Associated microRNA: miR-203a-5p
Biological function: promotes IL-1beta-induced chondrocytes apoptosis and OA progression; promotes inflammatory cytokine production; promotes ECM degradation
Molecular mechanism: ceRNA mechanism: circ-NFKB1 sponges miR-203a-5p to regulate ERBB4 expression
Biological pathway or process:

apoptosis (promotes); inflammation (promotes); ceRNA regulation (other)

Detected method:
Q
M
Validation methods:

Microarray; RT-qPCR; Clinical Sample Validation; RIP (RNA Immunoprecipitation); Luciferase Reporter Assay; Transfection; Annexin V/PI Flow Cytometry; ELISA; Western Blot; In Vivo Animal Model; H&E Staining; Bioinformatics Analysis

Clinical significance:

-

Description:

circ-NFKB1 is up-regulated in OA cartilage and IL-1beta-treated chondrocytes. It promotes OA-related chondrocyte apoptosis and inflammatory responses by sponging miR-203a-5p, thereby increasing ERBB4 expression; circ-NFKB1 knockdown alleviates OA phenotypes in vitro and in an ACLT rat OA model.

Confidence score:

0.7295

Other information
Title:

Circ-NFKB1 sponges miR-203a-5p to regulate ERBB4 expression and promotes IL-1beta induced chondrocytes apoptosis.

Journal: Journal of orthopaedic surgery and research
Published: 2023
PubMed ID: 37491357
Study type:

combined biological and clinical study

Data availability: GSE178724; GSE79258; GSE169077
Code availability: -