In exosomes released by primary mouse cardiomyocytes, circHIPK3 was increased after hypoxia pretreatment compared with normoxic exosomes.
Exosomal circHIPK3 Released from Hypoxia-Pretreated Cardiomyocytes Regulates Oxidative Damage in Cardiac Microvascular Endothelial Cells via the miR-29a/IGF-1 Pathway.